Parkinson’s disease (PD) is athat affects predominately dopamine-producing (“dopaminergic”) neurons in a specific area of the brain called .
Increasing dopamine levels is to increase the propbability of schizophrenia. Decreasing dopamine levels for schizophrenia is to cause Parkinsons. So, why not try ISF Neurofeedback + Photobiomodulation.
- Parkinson’s: When evolution leaves parts of us behind?
- Mitochondria and Parkinson’s Disease: Clinical, Molecular, and Translational Aspects
- Can neurofeedback improve and give you a better life? YES
- Can Neurofeedback curb parkinsons with early detection and therapy? Yes if you believe in neuroplasticity. Further to that, neurofeedback increases neuro networks and retrains that neuroplasticity by syncronising motor + memory + finer motor skills and more. Like they say, neurons that fire together stays together. Qeeg keeps them in check and Neurofeedback keeps them tuned towards optimal levels like.
Former USA Marine Jose Romero, How did you like your coffee, ambience ?
Can Photobiomodulation help?
Yes it can, simply by recharging your mitochondria
This functional threshold theory explains disease onset and progression better than the ascending spread theory for PD. https://www.sciencedirect.com/science/article/pii/S016622361630145X
the loss of the dopamine-producing cells in this area of the brain, accompanied by the presence of clumps of alpha-synuclein protein (known as Lewy bodies), has been the hallmark of Parkinson’s for decades.
Now that you know more, Do you want to know if Parkinsons is creeping through your back door?
The above link is just one of many. You may google the rest.
The QEEG brain map neurometric frequency readings identify areas of the brain with over or under-activity and if it is out of syncronization, and what training protocols to administer and to rescan again to precisely chart your progress. As such we can do a pre/post therapy scan that will correspond with your improvements.
Because we can identify and quantify which areas have abnormal activities, we can predict what type of symptoms are creeping into your backdoor and confirm what you may already be experiencing. For example, if grandparents becomes weak in eye hand coordination, balance, incongruent in speech, slowing down of basic motor skills or fine motor skills. QEEG will confirm what you may suspect or tell you if that was a one off slip and fall. QEEG identifies the location to train so that granny may walk better, talk better, remember better too.
More information: Braak’s 6 stage theory aims to describe how Parkinson’s spreads through the brain:
- Stage 1. Parkinson’s begins in the lower brainstem and the olfactory system. Changes in the areas of the brainstem could be responsible for some of the earliest symptoms of Parkinson’s, such as constipation. While changes in the olfactory system are believed to be responsible for the loss of sense of smell.
- Stage 2. Progression of Parkinson’s sees it move further up the brainstem, travelling to the areas below the substantia nigra that are involved in pain, sleep and mood.
- Stage 3. At the beginning of Stage 3, Parkinson’s has entered the substantia nigra and Lewy bodies begin to form.
- Stage 4. By this stage, a large proportion of the dopamine-producing cells have been affected. Additionally, Parkinson’s has continued to spread to the limbic system, which is involved in emotion, motivation and long term memory. At stage 4, significant damage has been done to the olfactory system.
- Stage 5. Parkinson’s has spread to the mesocortex — the area of the brain between the limbic system and outermost part of the brain known as the cerebral cortex. By this stage, Parkinson’s has begun to invade theneocortex, which is involved in higher-order brain functions such as perception, cognition and language. It spreads in all directions into the structures of the temporal, parietal, and frontal lobes.
- Stage 6. At this late stage of the condition, changes in the neocortex start to affect the memory and sensory areas in the brain.