Parkinson’s & Essential Tremors

What is Parkinsons? 

Parkinson’s disease (PD) is a neurodegenerative disorder that affects predominately dopamine-producing (“dopaminergic”) neurons in a specific area of the brain called substantia nigra.

Increasing dopamine levels is to increase the propbability of schizophrenia.  Decreasing dopamine levels for schizophrenia is to cause Parkinsons.  Try ISF Neurofeedback + Photobiomodulation. 

QEEG was is always recommendable fro a few reasons. 

  • Always good to know the full brain’s eletrical connectivity.
  • Always good to know if recommended medications have altered brainwave electrical patterns.  (hence, an eeg is done)
  • Since brain networks work together,  hence a Q+eeg is done to mathematically calculate the electrical activities.

Does photobiomodulation help improve Parkinson’s?



Photobiomodulation Therapy and Cell Therapy Improved Parkinson’s Diseases by Neuro-regeneration and Tremor Inhibition

1070nm penetrates deeper into the brain.  However 810nm is optimal.



Only Thinkfast

trains the cerebellum+full scalp + vagal area together.  
You cannot train what you cannot cover.
The cerebellum works with the rest of the brain together. 

Thinkfast in Black covers the areas.

Thinkfast (Black) Covers the cerebellum+whole scalp and vagal areas together.

Caution Note:  The light inside the white helmet does not reach what the shell covers.  

Photobiomodulation Solution!  

  1. Can neurofeedback improve and give you a better life?  YES
  2. Can Neurofeedback curb parkinsons with early detection and therapy?  Yes if you believe in neuroplasticity.  Further to that, neurofeedback increases neuro networks and retrains that neuroplasticity by syncronising motor + memory + finer motor skills and more.  Like they say, neurons that fire together stays together.   Qeeg keeps them in check and Neurofeedback keeps them tuned towards optimal levels like.

Can Photobiomodulation help?
Yes it can, simply by recharging your mitochondria

This functional threshold theory explains disease onset and progression better than the ascending spread theory for PD. 

Location of the substantia nigra. Created by FrozenManCC BY-SA 4.0   

the loss of the dopamine-producing cells in this area of the brain, accompanied by the presence of clumps of alpha-synuclein protein (known as Lewy bodies), has been the hallmark of Parkinson’s for decades.
Now that you know more, Do you want to know if Parkinsons is creeping through your back door? 

QEEG detects Parkinsons  – University of Oregon.

More information:  Braak’s 6 stage theory aims to describe how Parkinson’s spreads through the brain:

  • Stage 1. Parkinson’s begins in the lower brainstem and the olfactory system. Changes in the areas of the brainstem could be responsible for some of the earliest symptoms of Parkinson’s, such as constipation. While changes in the olfactory system are believed to be responsible for the loss of sense of smell.
  • Stage 2. Progression of Parkinson’s sees it move further up the brainstem, travelling to the areas below the substantia nigra that are involved in pain, sleep and mood.
  • Stage 3. At the beginning of Stage 3, Parkinson’s has entered the substantia nigra and Lewy bodies begin to form.
  • Stage 4. By this stage, a large proportion of the dopamine-producing cells have been affected. Additionally, Parkinson’s has continued to spread to the limbic system, which is involved in emotion, motivation and long term memory. At stage 4, significant damage has been done to the olfactory system.
  • Stage 5. Parkinson’s has spread to the mesocortex — the area of the brain between the limbic system and outermost part of the brain known as the cerebral cortex. By this stage, Parkinson’s has begun to invade theneocortex, which is involved in higher-order brain functions such as perception, cognition and language. It spreads in all directions into the structures of the temporal, parietal, and frontal lobes.
  • Stage 6. At this late stage of the condition, changes in the neocortex start to affect the memory and sensory areas in the brain.



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